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Seborrheic Dermatitis on the Scalp The Complete Guide

  • Is your scalp flaky, itchy, and red? You might be dealing with seborrheic dermatitis.
  • Confused about dandruff vs. seborrheic dermatitis? We clarify the differences.
  • Seeking effective treatments? Explore medical, home remedies, and barrier repair solutions.
  • Want long-term relief? Learn how to manage symptoms and improve scalp health.

This article is your in-depth guide to understanding and treating seborrheic dermatitis on the scalp. We’ll break down the complexities of this common condition, explore proven treatments, and empower you to make informed decisions for a healthier scalp. If you encounter unfamiliar terms or concepts, please ask in the comments below!

Image for Understanding Seborrheic Dermatitis of the Scalp

Understanding Seborrheic Dermatitis of the Scalp

Let’s start with the fundamentals of scalp seborrheic dermatitis. Knowing the basics is crucial for making informed treatment choices. If you’re already familiar with the condition, you can skip to the Treatments Section.

How Common is Scalp Seborrheic Dermatitis?

Seborrheic dermatitis affects a significant portion of the population, estimated at 3-5% [13]. This number can rise dramatically, reaching 40-50% in individuals with certain medical conditions like HIV/AIDS.

Scalp and Beyond: Areas Affected

The scalp is the most frequently affected area, where seborrheic dermatitis is often referred to as dandruff. However, “scalp seborrheic dermatitis” can be a more precise term, as the condition can extend beyond the scalp.

If your symptoms are limited to your scalp, consider yourself fortunate. Seborrheic dermatitis can become more persistent and aggressive when it spreads to other areas, including:

  • Nasolabial folds
  • Hairline
  • Ears
  • Eyebrows
  • Eyelids
  • Chin
  • Forehead

In rarer instances, symptoms can appear below the neckline, affecting the chest, upper back, and groin.

A key characteristic of these areas is their abundance of sebaceous glands, which secrete sebum (skin oil). Sebum plays a crucial role in the development of seborrheic dermatitis. The scalp is unique due to its high concentration of sebaceous glands and hair cover. While the impact of hair on seborrheic dermatitis isn’t fully understood, it definitely complicates treatment options.

Recognizing the Symptoms

Hallmark symptoms of seborrheic dermatitis include:

  • Skin flakes: Varying significantly in size, from fine to large and noticeable.
  • Dryness: The scalp may feel tight and dehydrated.
  • Inflammation and redness: Affected areas can appear pink or red.
  • Irritation and skin sensitivity: The scalp can become easily irritated and reactive.
  • Itch: A common and often bothersome symptom.
  • Excessive sebum production: While not always present, some experience increased scalp oiliness.

Seborrheic dermatitis is characterized by flare-ups, with symptoms fluctuating in intensity. Sometimes they subside, and other times they erupt and become difficult to manage.

Why Accurate Diagnosis Matters

If you suspect you have seborrheic dermatitis but aren’t certain, getting a confirmed diagnosis is essential. Accurate diagnosis is the cornerstone of effective management. Treating the wrong condition can waste time and money on ineffective treatments.

Image for Unraveling the Causes of Seborrheic Dermatitis

Unraveling the Causes of Seborrheic Dermatitis

Despite extensive research, the exact cause of seborrheic dermatitis remains unclear. However, the prevailing theory points to a combination of factors. Ongoing advancements in genetic sequencing may offer clearer insights in the future, but for now, the current working theory provides the best explanation.

This theory centers around the interaction of three key elements:

  • Malassezia yeasts: A type of fungus naturally found on the skin.
  • Sebaceous gland secretions (sebum): Skin oil that provides nourishment for Malassezia.
  • Individual susceptibility: Unique factors in each person that influence their reaction to Malassezia and sebum.

These three factors are consistently highlighted in medical literature regarding seborrheic dermatitis.


The Skin Microflora: A Broader Perspective
Emerging research suggests that the overall skin microflora, the complex community of microorganisms on our skin, may have a more significant role than Malassezia alone. We’ll delve into this later.

Malassezia Yeasts: The Prime Suspect

Malassezia yeasts have been the focus of much research. They are considered commensal organisms, meaning they normally live on human skin without causing harm. However, under certain conditions, they can become pathogenic and contribute to conditions like pityriasis versicolor and Malassezia folliculitis [1].

In seborrheic dermatitis, the role of Malassezia is less straightforward. While they don’t appear to transform into their pathogenic hyphae form [2], they are still implicated in the condition’s development.

The link is largely based on the observation that antifungal treatments often lead to rapid symptom improvement. This is thought to be due to the reduction of Malassezia activity on the skin surface. This connection has led decades of research to conclude that these yeasts play a critical role [4].

Currently, the primary understanding is that an overgrowth of Malassezia yeasts is central to seborrheic dermatitis. Reducing Malassezia activity typically leads to symptom relief.


Malassezia Species: Could Specific Types Matter?
Studies comparing the skin microflora of individuals with and without seborrheic dermatitis have revealed differences in the proportions of specific Malassezia species [3]. This suggests that certain species might be more involved in the condition.

Oleic Acid and Scalp Irritation

Malassezia yeasts rely on lipids (fats and oils) for their growth, and sebum, rich in lipids, provides an ideal environment and nutrient source. As Malassezia breaks down sebum, it produces byproducts, including enzymes, free fatty acids, and phospholipases. One notable byproduct is oleic acid in its free fatty acid form.

Intriguingly, even when Malassezia yeasts are reduced with antifungal pretreatment, applying oleic acid directly to the skin of susceptible individuals can trigger irritation, disrupt the skin barrier, and cause symptoms identical to seborrheic dermatitis and dandruff [4, 5, 6].

This suggests that the symptoms of seborrheic dermatitis might be caused by oleic free fatty acids, a byproduct of Malassezia activity, rather than the yeasts themselves. In this scenario, Malassezia’s role is contributory, producing oleic acid on the skin’s surface.

The Crucial Role of a Healthy Skin Barrier

Your skin acts as a vital protective barrier against the external environment. This barrier, the outermost layer of your skin, employs various mechanisms to defend against foreign invaders and antigens.

In individuals with seborrheic dermatitis, this skin barrier is often compromised [7]. This allows external irritants to penetrate deeper skin layers, leading to increased irritation, inflammation, and also contributing to moisture loss and dryness.

While oleic acid can directly damage the skin barrier, individual differences in barrier function likely determine who develops seborrheic dermatitis. A robust skin barrier should neutralize topical irritants effectively, preventing the skin’s immune system from overreacting.

Recent Insights: The Microbial World

Beyond Fungi: The Bacterial Connection

While research has traditionally focused on the fungal aspect (Malassezia), recent investigations highlight the potential significance of bacteria in seborrheic dermatitis [8, 9].

Overall skin micrflora is likely to play a more important role in seborrheic dermatitis than malassezia alone

Key findings from these studies include:

  • Bacteria and Dandruff Severity: The types of bacteria on the skin show a stronger correlation with dandruff severity than fungi [10].
  • Combined Microbial Impact: Both bacteria and fungi are linked to dandruff and seborrheic dermatitis, but skin symptoms appear more closely related to the bacterial flora [11].
  • Microbial Shift: Changes in the entire skin microflora are more strongly associated with seborrheic dermatitis than just Malassezia populations [12].

These findings suggest that future research may uncover new treatment approaches and long-term management strategies targeting the broader skin microflora.

Image for Scalp Seborrheic Dermatitis: Unique Characteristics

Scalp Seborrheic Dermatitis: Unique Characteristics

Seborrheic dermatitis on the scalp has distinct features that influence its development and treatment. Understanding these nuances can provide valuable insights and improve your chances of successful management.

Dandruff vs. Seborrheic Dermatitis: Are They Different?

The terms “seborrheic dermatitis” and “dandruff” are often used interchangeably when referring to the scalp. Most medical literature considers them to be the same condition, with the main distinction being that dandruff is limited to the scalp, while seborrheic dermatitis can occur elsewhere [13].

However, some researchers propose a further differentiation:

  • Dandruff: Milder symptoms, limited in severity.
  • Seborrheic Dermatitis: A more aggressive form of dandruff.

While not universally accepted, this distinction may be helpful. Separating them could improve our understanding of how dandruff progresses to more severe seborrheic dermatitis and spreads beyond the scalp. It’s possible that dandruff is an early stage of seborrheic dermatitis, acting as an initial warning sign.

Clinical Distinctions

Some experts even describe specific clinical differences [14]:

Visual representation of key differences between dandruff and seborrheic dermatitis

Dandruff:

  • Characterized by scales and subclinical inflammation (inflammation that’s very difficult to detect), restricted to the scalp. More specifically, it shows “scattered presence of lymphoid cells and squirting capillaries in the papillary dermis, hints of spongiosis and focal parakeratosis.”

Seborrheic Dermatitis:

  • Characterized by scales, significant inflammation, often extending beyond the scalp. More specifically, it presents as “spongiotic dermatitis in which mounds of parakeratosis and scale crusts form at lips of infundibular ostia.”

While the more detailed descriptions can be complex, the key takeaway is that scalp seborrheic dermatitis can be viewed as a more intense form of dandruff with inflammation as a primary feature.

Hair’s Impact on Seborrheic Dermatitis

Hair follicles have been suggested as potential reservoirs for drug delivery, potentially enhancing the effectiveness of topical treatments [15]. This would imply that treatments might work better in hair-bearing areas.

However, those with seborrheic dermatitis know that hair often makes the condition harder to manage. A 1993 review even suggested that shaving affected areas can significantly improve or even resolve symptoms [16].

Unfortunately, shaving the scalp isn’t a practical solution for most. We must acknowledge the challenges hair presents and adjust treatment strategies accordingly.

Hair Products and Irritation

Some researchers have proposed that scalp seborrheic dermatitis might arise without microbial involvement, simply as a reaction to irritating hair care products or environmental substances [17].

For some, switching hair products might be enough to trigger and maintain remission. If you explore this route, compare product ingredient lists carefully, focusing on the first 3-5 ingredients, as these are present in the highest concentrations. However, be aware that many products share similar base ingredients.

The Role of Heat

Heat might also play a role in seborrheic dermatitis. Thermal imaging studies indicate that areas commonly affected by seborrheic dermatitis, particularly on the face, tend to have higher skin temperatures:

  • [Relation Between Skin Temperature and Location of Facial Lesions in Seborrheic Dermatitis][1]

One theory suggests that increased skin temperature could lead to increased Malassezia activity and, consequently, more oleic free fatty acid production. Alternatively, it’s possible that the high density of sebaceous glands in these areas leads to increased blood flow and higher skin temperature, making temperature a secondary factor.

Hair Loss and Seborrheic Dermatitis

Hair loss isn’t directly a symptom of scalp seborrheic dermatitis or dandruff. However, if these conditions become severe and are left unchecked, hair loss can become a concern.

This is often due to:

  • Physical damage: Scratching and picking at the scalp.
  • Hair follicle blockage: Flakes and scales can clog hair follicles.
  • Inflammation: Chronic inflammation can disrupt hair growth cycles.

These factors can weaken hair, making it thinner and more prone to breakage [], leading to noticeable hair loss.

One study showed that individuals with dandruff lost significantly more hair (100-300 hairs daily) compared to those without (50-100 hairs daily) [pubb id=”18489295″]. This suggests that seborrheic dermatitis can lead to 2-6 times more hair shedding than normal.

Fortunately, many seborrheic dermatitis treatments can reduce hair loss and promote healthier hair growth. Addressing the scalp condition often resolves associated hair loss issues. This is discussed further in this article: Reversing Seborrheic Dermatitis and Hair Loss.

Image for Treatment Strategies: A Two-Pronged Approach

Treatment Strategies: A Two-Pronged Approach

Given the multifaceted nature of seborrheic dermatitis, effective treatment should address two key aspects:

  • Reducing Malassezia and oleic acid levels.
  • Improving and restoring the skin barrier function.

While targeting just one of these can provide some relief, a combined approach is more likely to achieve long-term success.

Two primary treatment approaches to scalp seborrheic dermatitis - malassezia control and barrier restoration

1. Irritant Reduction: Antifungal Treatments

Based on the theory that Malassezia byproducts trigger symptoms, antifungal agents are a primary treatment for scalp seborrheic dermatitis. They reduce Malassezia activity and often provide rapid relief.

The action pathway is:

Antifungal → Reduced Malassezia → Reduced Oleic Acid → Symptom Relief

However, some evidence suggests that antifungals might work differently than simply reducing Malassezia. While they effectively suppress Malassezia in lab settings [19, 20, 21], their impact on skin surface Malassezia counts in real-world use is often minimal []. It’s possible they provide relief by influencing fungal/host gene expression or affecting the inflammatory process.

2. Skin Barrier Restoration

As discussed earlier, compromised barrier function is a significant factor in seborrheic dermatitis. While systemic approaches like diet and stress management can help, specially formulated topical products can also improve barrier function and prevent further disruption.

Strengthening the skin barrier can reduce the irritant effects of Malassezia and oleic acid, leading to gradual symptom improvement.

The action pathway is:

Barrier Repair → Reduced Sensitivity → Lack of Symptoms

This approach aims for longer-term relief by enhancing the skin’s natural defenses rather than just eliminating Malassezia, which is a normal skin inhabitant. While barrier repair strategies are still evolving, they hold significant promise for the future.

Image for Additional Tips for Treatment Success

Additional Tips for Treatment Success

Regardless of your treatment approach, these factors can improve outcomes:

Extended Application Time

Leaving shampoo on longer can improve effectiveness [59]. Piroctone olamine benefits more from longer exposure than ketoconazole, but even ketoconazole shows some improvement.

However, prolonged surfactant exposure can disrupt the skin barrier [60]. Longer-term studies are needed to fully assess the impact of extended exposure times.

Cooler Water Temperatures

Lower water temperatures during washing can reduce irritation []. Surfactants can be harsh, and minimizing irritation at each wash is helpful. Consider washing hair with cool or cold water.

Image for Author's Perspective

Author’s Perspective

Seborrheic dermatitis profoundly impacted my life when it spread beyond my scalp to my face. It affected my social life and caused considerable distress.

After years of experimentation, I found solutions and achieved remission. My interest in the root causes remains.

My understanding continues to evolve, but I believe systemic factors are key:

  • Metabolic issues (especially fat metabolism)
  • Lifestyle (sunlight, location, stress)
  • Diet
  • Hormones
  • Disease states

These factors influence immune stability, systemic inflammation, microbiota, and lipid oxidation, leading to an unstable skin microflora, impaired barrier function, and over-reactivity to irritants.

Topical treatments manage symptoms, but addressing underlying issues is crucial for long-term remission and overall health. This guide focuses on topical treatments. Underlying issues are discussed further here: Seborrheic Dermatitis – The Owner’s Manual.

Image for Summary: Key Takeaways

Summary: Key Takeaways


1. Seborrheic dermatitis is a common, but not fully understood, skin condition affecting sebum-rich areas like the scalp and face.
2. Dandruff and seborrheic dermatitis are often used interchangeably, with seborrheic dermatitis potentially being a more severe form of dandruff.
3. The condition likely results from interactions between Malassezia yeasts, sebum, and individual susceptibility.
4. Hair can complicate scalp seborrheic dermatitis management, and hair loss can be an indirect consequence.
5. Antifungal agents (zinc pyrithione, ketoconazole, selenium sulfide, coal tar, tea tree oil) are the most common treatments.
6. Scalp treatment primarily uses antifungal shampoos and conditioners.
7. Apple cider vinegar, raw honey, and Dead Sea salt are popular home remedies.
8. Improving barrier function through gentle hair washing may be beneficial.

We are all navigating this together. Share your experiences or questions in the comments below.

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References

  1. E Guého, T Boekhout, H R Ashbee, J Guillot, A Van Belkum, J Faergemann "The role of Malassezia species in the ecology of human skin and as pathogens." Medical mycology 36 Suppl 1 (1999): 220-9. PubMed
  2. Robert A Schwartz, Christopher A Janusz, Camila K Janniger "Seborrheic dermatitis: an overview." American family physician 74.1 (2006): 125-30. PubMed
  3. A Nakabayashi, Y Sei, J Guillot "Identification of Malassezia species isolated from patients with seborrhoeic dermatitis, atopic dermatitis, pityriasis versicolor and normal subjects." Medical mycology 38.5 (2001): 337-41. PubMed
  4. R J Hay "Malassezia, dandruff and seborrhoeic dermatitis: an overview." The British journal of dermatology 165 Suppl 2 (2012): 2-8. PubMed
  5. Yvonne M DeAngelis, Christina M Gemmer, Joseph R Kaczvinsky, Dianna C Kenneally, James R Schwartz, Thomas L Dawson "Three etiologic facets of dandruff and seborrheic dermatitis: Malassezia fungi, sebaceous lipids, and individual sensitivity." The journal of investigative dermatology. Symposium proceedings / the Society for Investigative Dermatology, Inc. [and] European Society for Dermatological Research 10.3 (2005): 295-7. PubMed
  6. E Boelsma, H Tanojo, H E Boddé, M Ponec "Assessment of the potential irritancy of oleic acid on human skin: Evaluation in vitro and in vivo." Toxicology in vitro : an international journal published in association with BIBRA 10.6 (2012): 729-42. PubMed
  7. James R Schwartz, Andrew G Messenger, Antonella Tosti, Gail Todd, Maria Hordinsky, Roderick J Hay, Xuemin Wang, Claus Zachariae, Kathy M Kerr, James P Henry, Rene C Rust, Michael K Robinson "A comprehensive pathophysiology of dandruff and seborrheic dermatitis – towards a more precise definition of scalp health." Acta dermato-venereologica 93.2 (2013): 131-7. PubMed
  8. Mami Tajima, Takashi Sugita, Akemi Nishikawa, Ryoji Tsuboi "Molecular analysis of Malassezia microflora in seborrheic dermatitis patients: comparison with other diseases and healthy subjects." The Journal of investigative dermatology 128.2 (2008): 345-51. PubMed
  9. Funda Tamer, Mehmet Eren Yuksel, Evren Sarifakioglu, Yavuz Karabag "is the most common bacterial agent of the skin flora of patients with seborrheic dermatitis." Dermatology practical & conceptual 8.2 (2018): 80-84. PubMed
  10. Zhijue Xu, Zongxiu Wang, Chao Yuan, Xiaoping Liu, Fang Yang, Ting Wang, Junling Wang, Kenji Manabe, Ou Qin, Xuemin Wang, Yan Zhang, Menghui Zhang "Dandruff is associated with the conjoined interactions between host and microorganisms." Scientific reports 6 (2016): 24877. PubMed
  11. Taehun Park, Hye-Jin Kim, Nu Ri Myeong, Hyun Gee Lee, Ilyoung Kwack, Johnhwan Lee, Beom Joon Kim, Woo Jun Sul, Susun An "Collapse of human scalp microbiome network in dandruff and seborrhoeic dermatitis." Experimental dermatology 26.9 (2018): 835-838. PubMed
  12. Luciana Campos Paulino "New perspectives on dandruff and seborrheic dermatitis: lessons we learned from bacterial and fungal skin microbiota." European journal of dermatology : EJD 27.S1 (2018): 4-7. PubMed
  13. C Piuerard-Franchimont, E Xhauflaire-Uhoda, G E Piuerard "Revisiting dandruff." International journal of cosmetic science 28.5 (2008): 311-8. PubMed
  14. C Piuerard-Franchimont, J F Hermanns, H Degreef, G E Piuerard "From axioms to new insights into dandruff." Dermatology (Basel, Switzerland) 200.2 (2000): 93-8. PubMed
  15. J Lademann, H Richter, U F Schaefer, U Blume-Peytavi, A Teichmann, N Otberg, W Sterry "Hair follicles – a long-term reservoir for drug delivery." Skin pharmacology and physiology 19.4 (2006): 232-6. PubMed
  16. A Rebora, F Rongioletti "The red face: seborrheic dermatitis." Clinics in dermatology 11.2 (1993): 243-51. PubMed
  17. Rekha A. Sheth, Sharat C. Desai "Dandruff: Assessment and Management" Wiley 22.9 (2008): 511-514. doi.org
  18. Rodney D Sinclair, James R Schwartz, Heather L Rocchetta, Thomas L Dawson, Brian K Fisher, Knut Meinert, Elizabeth A Wilder "Dandruff and seborrheic dermatitis adversely affect hair quality." European journal of dermatology : EJD 19.4 (2009): 410-1. PubMed
  19. Takashi Sugita, Mami Tajima, Tomonobu Ito, Masuyoshi Saito, Ryoji Tsuboi, Akemi Nishikawa "Antifungal activities of tacrolimus and azole agents against the eleven currently accepted Malassezia species." Journal of clinical microbiology 43.6 (2005): 2824-9. PubMed
  20. A K Gupta, Y Kohli, A Li, J Faergemann, R C Summerbell "In vitro susceptibility of the seven Malassezia species to ketoconazole, voriconazole, itraconazole and terbinafine." The British journal of dermatology 142.4 (2000): 758-65. PubMed
  21. Karla Carvalho Miranda, Crystiane Rodrigues de Araujo, Carolina Rodrigues Costa, Xisto Sena Passos, Orionalda de Fátima Lisboa Fernandes, Maria do Rosário Rodrigues Silva "Antifungal activities of azole agents against the Malassezia species." International journal of antimicrobial agents 29.3 (2007): 281-4. PubMed
  22. Luis J Borda, Tongyu C Wikramanayake "Seborrheic Dermatitis and Dandruff: A Comprehensive Review." Journal of clinical and investigative dermatology 3.2 (2017). PubMed
  23. G C Priestley, J C Brown "Acute toxicity of Zinc pyrithione to human skin cells in vitro." Acta dermato-venereologica 60.2 (1980): 145-48. PubMed
  24. Claudine Piuerard-Franchimont, Vueronique Goffin, Jacques Decroix, Guerald E Piuerard "A multicenter randomized trial of ketoconazole 2% and zinc pyrithione 1% shampoos in severe dandruff and seborrheic dermatitis." Skin pharmacology and applied skin physiology 15.6 (2002): 434-41. PubMed
  25. A C Bulmer, G S Bulmer "The antifungal action of dandruff shampoos." Mycopathologia 147.2 (2000): 63-5. PubMed
  26. F W Danby, W S Maddin, L J Margesson, D Rosenthal "A randomized, double-blind, placebo-controlled trial of ketoconazole 2% shampoo versus selenium sulfide 2.5% shampoo in the treatment of moderate to severe dandruff." Journal of the American Academy of Dermatology 29.6 (1993): 1008-12. PubMed
  27. Goldenberg Gary "Optimizing treatment approaches in seborrheic dermatitis." The Journal of clinical and aesthetic dermatology 6.2 (2013): 44-9. PubMed
  28. Tanya M Barnes, Kerryn A Greive "Topical pine tar: History, properties and use as a treatment for common skin conditions." The Australasian journal of dermatology 58.2 (2018): 80-85. PubMed
  29. M H Schmid, H C Korting "Coal tar, pine tar and sulfonated shale oil preparations: comparative activity, efficacy and safety." Dermatology (Basel, Switzerland) 193.1 (1997): 1-5. PubMed
  30. P Nenoff, U F Haustein, A Fiedler "The antifungal activity of a coal tar gel on Malassezia furfur in vitro." Dermatology (Basel, Switzerland) 191.4 (1996): 311-4. PubMed
  31. Aditya K Gupta, S E Madzia, Roma Batra "Etiology and management of Seborrheic dermatitis." Dermatology (Basel, Switzerland) 208.2 (2004): 89-93. PubMed
  32. Judith H J Roelofzen, Katja K H Aben, Pieter G M van der Valk, Jeanette L M van Houtum, Peter C M van de Kerkhof, Lambertus A L M Kiemeney "Coal tar in dermatology." The Journal of dermatological treatment 18.6 (2008): 329-34. PubMed
  33. Kapila V Paghdal, Robert A Schwartz "Topical tar: back to the future." Journal of the American Academy of Dermatology 61.2 (2009): 294-302. PubMed
  34. M R Pittelkow, H O Perry, S A Muller, W Z Maughan, P C O'Brien "Skin cancer in patients with psoriasis treated with coal tar. A 25-year follow-up study." Archives of dermatology 117.8 (1981): 465-8. PubMed
  35. David Larson, Sharon E Jacob "Tea tree oil." Dermatitis : contact, atopic, occupational, drug 23.1 (2013): 48-9. PubMed
  36. K A Hammer, C F Carson, T V Riley "In vitro susceptibility of Malassezia furfur to the essential oil of Melaleuca alternifolia." Journal of medical and veterinary mycology : bi-monthly publication of the International Society for Human and Animal Mycology 35.5 (1998): 375-7. PubMed
  37. Andrew C Satchell, Anne Saurajen, Craig Bell, Ross StC Barnetson "Treatment of dandruff with 5% tea tree oil shampoo." Journal of the American Academy of Dermatology 47.6 (2002): 852-5. PubMed
  38. E L HAZEN, R BROWN "Fungicidin, an antibiotic produced by a soil actinomycete." Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.) 76.1 (2004): 93-7. PubMed
  39. E L HAZEN, R BROWN "Two antifungal agents produced by a soil actinomycete." Science (New York, N.Y.) 112.2911 (2004): 423. PubMed
  40. R Lorenzini, R Mercantini, F De Bernardis "In vitro sensitivity of Malassezia spp. to various antimycotics." Drugs under experimental and clinical research 11.6 (1986): 393-5. PubMed
  41. M Buslau, H Hänel, H Holzmann "[The significance of yeasts in seborrheic eczema]." Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete 40.10 (1990): 611-3. PubMed
  42. Buenigne-Ernest Amborabue, Pierrette Fleurat-Lessard, Jean-Franueois Chollet, Gabriel Roblin "Antifungal effects of salicylic acid and other benzoic acid derivatives towards Eutypa lata: structure–activity relationship" Elsevier BV 40.12 (2003): 1051-1060. doi.org
  43. L Chao "Simultaneous determination of four anti-dandruff agents including octopirox in shampoo products by reversed-phase liquid chromatography." International journal of cosmetic science 23.3 (2012): 183-8. PubMed
  44. R A Squire, K Goode "A randomised, single-blind, single-centre clinical trial to evaluate comparative clinical efficacy of shampoos containing ciclopirox olamine (1.5%) and salicylic acid (3%), or ketoconazole (2%, Nizoral) for the treatment of dandruff/seborrhoeic dermatitis." The Journal of dermatological treatment 13.2 (2002): 51-60. PubMed
  45. M Lodén, C Wessman "The antidandruff efficacy of a shampoo containing piroctone olamine and salicylic acid in comparison to that of a zinc pyrithione shampoo." International journal of cosmetic science 22.4 (2012): 285-9. PubMed
  46. M Charach "Malassezia dermatitis." The Canadian veterinary journal = La revue veterinaire canadienne 38.5 (1997): 311-4. PubMed
  47. A Baroni, R De Rosa, A De Rosa, G Donnarumma, P Catalanotti "New strategies in dandruff treatment: growth control of Malassezia ovalis." Dermatology (Basel, Switzerland) 201.4 (2001): 332-6. PubMed
  48. Tahereh Eteraf-Oskouei, Moslem Najafi "Traditional and modern uses of natural honey in human diseases: a review." Iranian journal of basic medical sciences 16.6 (2013): 731-42. PubMed
  49. Bruno Burlando, Laura Cornara "Honey in dermatology and skin care: a review." Journal of cosmetic dermatology 12.4 (2013): 306-13. PubMed
  50. N S Al-Waili "Therapeutic and prophylactic effects of crude honey on chronic seborrheic dermatitis and dandruff." European journal of medical research 6.7 (2001): 306-8. PubMed
  51. S E Blair, N N Cokcetin, E J Harry, D A Carter "The unusual antibacterial activity of medical-grade Leptospermum honey: antibacterial spectrum, resistance and transcriptome analysis." European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology 28.10 (2009): 1199-208. PubMed
  52. N S Al-Waili "An alternative treatment for pityriasis versicolor, tinea cruris, tinea corporis and tinea faciei with topical application of honey, olive oil and beeswax mixture: an open pilot study." Complementary therapies in medicine 12.1 (2004): 45-7. PubMed
  53. Ehrhardt Proksch, Hans-Peter Nissen, Markus Bremgartner, Colin Urquhart "Bathing in a magnesium-rich Dead Sea salt solution improves skin barrier function, enhances skin hydration, and reduces inflammation in atopic dry skin." International journal of dermatology 44.2 (2005): 151-7. PubMed
  54. S Halevy, H Giryes, M Friger, N Grossman, Z Karpas, B Sarov, S Sukenik "The role of trace elements in psoriatic patients undergoing balneotherapy with Dead Sea bath salt." The Israel Medical Association journal : IMAJ 3.11 (2001): 828-32. PubMed
  55. F Levi-Schaffer, J Shani, Y Politi, E Rubinchik, S Brenner "Inhibition of proliferation of psoriatic and healthy fibroblasts in cell culture by selected Dead-sea salts." Pharmacology 52.5 (1996): 321-8. PubMed
  56. Emmilia Hodak, Alice B Gottlieb, Tsvi Segal, Yael Politi, Lea Maron, Jaqueline Sulkes, Michael David "Climatotherapy at the Dead Sea is a remittive therapy for psoriasis: combined effects on epidermal and immunologic activation." Journal of the American Academy of Dermatology 49.3 (2003): 451-7. PubMed
  57. Jana Kazandjieva, Ivan Grozdev, Razvigor Darlenski, Nikolai Tsankov "Climatotherapy of psoriasis." Clinics in dermatology 26.5 (2009): 477-85. PubMed
  58. Clive R Harding, Jane R Matheson, Michael Hoptroff, David A Jones, Yanjun Luo, Fiona L Baines, Shengjun Luo "A high glycerol-containing leave-on scalp care treatment to improve dandruff." Skinmed 12.3 (2014): 155-61. PubMed
  59. C Piérard-Franchimont, E Uhoda, G Loussouarn, D Saint-Léger, G E Piérard "Effect of residence time on the efficacy of antidandruff shampoos." International journal of cosmetic science 25.6 (2010): 267-71. PubMed
  60. K P Ananthapadmanabhan, David J Moore, Kumar Subramanyan, Manoj Misra, F Meyer "Cleansing without compromise: the impact of cleansers on the skin barrier and the technology of mild cleansing." Dermatologic therapy 17 Suppl 1 (2004): 16-25. PubMed
  61. E Berardesca, G P Vignoli, F Distante, P Brizzi, G Rabbiosi "Effects of water temperature on surfactant-induced skin irritation." Contact dermatitis 32.2 (1995): 83-7. PubMed
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About Michael Anders

After being affected by seborrheic dermatitis, I have made it my goal to gather and organize all the information that has helped me in my journey.

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